This morning started early after a long day yesterday. The burning sensation disappeared. I’m pretty sure it was something in the bath oil that caused me to have a bad reaction.
I drove to Yosemite (it’s an hour away) via the one-way bridge that cars cross in both directions. I got into the Yosemite gate pretty quickly. I drove another 20 minutes to the meeting spot. I was early so I stopped to take some photos at some take-out points.
I met up with Katherine and we got in her car to go to the next spot. It was Tuolumne Grove to see the gigantic majestic redwood trees. We walk through the forest.
Then we got in the car and drove to Olmsted Point where we walked a bit, took pictures and meditated for a while. There’s a fire in the park, so today the air was smoky.
Then we went to Taneya Lake where we ate a packed lunch she brought. It was nice to sit in her camp chairs and have lunch by a beautiful lake. One interesting fact: the boulders and trees all had rings around them. It was a trail left by the lake this year after the historic snowfall. The water rose so high!
Then we went to Tuolumne Meadow to Soda Springs. It’s a nice walk to see water that is actually naturally carbonated. Scientists can’t figure out why. I also used an outhouse for the first time in a long time.
We then headed back to Olmsted Point because the smoke cleared and I was able to get a better photo.
That was the end of my tour/walk for the day. I drove back to the room to shower and get ready for dinner. Tonight I decided to get take out and eat on my porch.
Now I have to go to bed, because tomorrow is coming soon. It was going to rain, so this walk will be interesting!
By the way, if you’re interested in the dog, she was caught watching football with her boyfriend.
Most of us know from experience that tossing and turning at night means you’ll feel bad the next day. And many of us understand that there are long-term health consequences when we get too little sleep.
But recently I discovered something new from a fascinating study into sleep and its effect on how quickly women’s genes age after menopause – what’s known as their ‘epigenetic age’. (Carroll et al. 2017)
In the study, postmenopausal women with five insomnia symptoms were biologically almost two years older than comparable women without insomnia symptoms. The more symptoms of insomnia – Waking up during the night, not being able to fall asleep, disturbed sleep and so on – the greater the effect. However, if you only sleep six hours a night, it appears that your epigenetic age does not increase, as long as it is a good night’s sleep. And that, of course, is what many of us have problems with.
So what can we do about this? I would like to point out that the research shows that our genes are not completely out of our control, and that we can influence how they respond by taking better care of ourselves. For those of us who have gone through menopause and may be having trouble sleeping, this means looking at ways we can improve our rest.
4 sleep tips to try
Keep a regular schedule. Your brain likes consistency, so go to bed at 11 p.m. and at the same regular time every night and in a dark room. When you get up, immediately soak up the morning light by going outside or opening a window. Ensuring good light health is key to your circadian health.
Turn off the electronics! Computers, TVs, phones: they’re all the enemy of sleep. Even a short duration of light shining into your eyes tricks your brain into thinking it’s time to wake up. Every night at least an hour before bed, turn off the phone and put it on the charger in another room so you aren’t tempted to look at it when you can’t sleep.
Eat early and in moderation. Heavy meals just before bed disrupt sleep, while avoiding caffeine and stimuli (such as dramatic/stressful TV shows) can also help with sleep.
Once you’re in bed, stay relaxed. Once you’re in bed, consider practicing relaxing belly breathing or guided visualizations like that of Yoga Nidra.
In the checklist above you will see some of my favorite sleep remedies, including of course L-Tryptophan, which can be combined with magnesium for an even more relaxing effect.
Reference: Carroll, JE et al. 2017. Epigenetic aging and immune aging in women with insomnia symptoms: findings from the Women’s Health Initiative Study, Biological Psychiatry 81(2):136-144.
I am Dr. Susan E. Brown. I am a clinical nutritionist, medical anthropologist, writer and motivational coach speaker. Learn my proven 6-step natural approach to bone health in my online courses.
Injuries to the anterior cruciate ligament (ACL), located in the knee, are commonly thought to be caused by acute traumatic events, such as sudden twists. Led by researchers at Penn State, new work analyzing an animal model of ACLs suggests that such injuries may also occur as a result of chronic overuse, specifically due to a reduced ability to repair microtraumas associated with overuse. Importantly, according to the team, women are less able to heal from these microtraumas than men, which could explain why women are two to eight times more likely to tear their ACL ligaments than men.
“ACL tears are one of the most common injuries, affecting more than 200,000 people in the US each year, and women are known to be particularly susceptible,” said lead researcher Spencer Szczesny, associate professor of biomedical engineering and orthopedics and rehabilitation at Penn . Stands. “Although recent research suggests that chronic overuse may lead to ACL injuries, no one has yet examined the different biological responses of female and male ACLs to applied force.”
From the research, published in the Journal of Orthopedic Researchresearchers placed ACLs from deceased male and female rabbits in a custom-built bioreactor that simulated the conditions of a living animal but allowed for direct observation and measurement of the tissue. They then applied repetitive forces to the ACLs that mimicked the forces that would naturally occur during activities such as standing, walking and trotting, and measured the expression of genes related to healing.
In male samples, the team found that low and moderate applied forces, such as those that would occur while standing or walking, resulted in increased expression of anabolic genes, which are related to building molecules needed for healing. In contrast, greater applied forces, such as would occur with repetitive trotting, reduced the expression of these anabolic genes. However, in female samples, the amount of force applied did not affect the level of anabolic gene expression.
“It didn’t matter whether there was low, medium or high activity in women,” said Lauren Paschall, a biomedical engineering graduate student at Penn State and first author of the paper. “Female ACLs exposed to chronic use simply did not heal as well as male ACLs, which may explain why women are prone to injuries. This supports the hypothesis that non-contact ACL injuries are attributed to microtraumas associated with chronic overuse that predisposes the ACL make for injuries.” .”
According to the researchers, one explanation for the gender differences the team observed could be due to the higher amounts of estrogen in women.
“Some studies have shown that the overall effect of estrogen on ACL injury is negative,” Paschall said. “Specifically, studies have shown that human women are more likely to tear their ACLs during the preovulatory phase, when estrogen levels are high, than during the postovulatory phase, when estrogen levels are low.”
She said the team plans to further investigate the role of estrogen in ACL injuries.
Szczesny noted that although the team’s research did not take place in humans, the findings may indicate that providing women with additional recovery time after injuries could be beneficial.
“Ultimately, this work could also help identify targets for therapies to prevent ACL injury in women,” he said.
Other Penn State authors on the paper include Sabrina Carrozzi, graduate student; Erdem Tabdanov, assistant professor of pharmacology; and Aman Dhawan, professor of orthopedics and rehabilitation.
The Orthopedic Research and Education Foundation and the Congressional Directed Medical Research Program supported this research.
The information shared above is for informational purposes only and is not intended as medical or nutritional therapy advice; it does not diagnose, treat or cure any disease or condition; it should not be used as a substitute or substitute for medical advice from physicians and trained medical professionals. If you are under the care of a healthcare professional or are currently taking prescription medications, you should discuss any changes in your diet and lifestyle or possible use of nutritional supplements with your doctor. You should not stop prescribed medications without first consulting your doctor.
Osteoarthritis is the most common joint disease in the United States and can cause pain, stiffness, and limitation in daily activities. Unfortunately, many people feel forced to give up their favorite sport or lifestyle as a result of being labeled “osteoarthritis.”
A diagnosis of ‘osteoarthritis’ can be confusing.
What does it mean? Does this mean that you cannot cycle, walk, hike or exercise without pain? How should you treat it and what can you do to prevent it from getting worse?
What is osteoarthritis?
Osteoarthritis, also known as degenerative joint disease, occurs when the cartilage that covers the ends of the bones in your joints gradually deteriorates. The degenerative process, which typically affects weight-bearing joints such as the knees, hips and hands, causes thinning of the cartilage, narrowing of joint spaces and reduced cushioning between the bones. This breakdown process puts more pressure on the sensitive surface of the bones in the joint, causing inflammation, pain, bone spurs and further wear and tear of the joint.
Being diagnosed with osteoarthritis does NOT mean that you cannot or should not have an active lifestyle. On the contrary, activity can actually improve the symptoms of osteoarthritis and slow or stop its progression. Many people with osteoarthritis lead an active, healthy lifestyle, without restrictions.
How is osteoarthritis diagnosed?
Osteoarthritis affects more than 32.5 million Americans and is usually diagnosed by physical examination and an X-ray. Based on the X-ray images, doctors will look for a smaller distance between the bones in the joint and for signs of inflammation. They will look at how much cartilage there is in the joint to protect the underlying bone from further wear and tear. They will also look for bone fragments or ‘spurs’ that are sticking into the joint and could cause more pain.
X-ray findings should be compared with a physical examination to assess actual loss of range of motion, stiffness, weakness, and activity limitations before determining a treatment plan.
Did you know that there are people with osteoarthritis who are NOT symptomatic? They have no pain, stiffness, swelling, or disability associated with the condition. In fact, they may not even know they have it!
Evidence of degenerative joint changes can be seen in young adults to older adults, but the risk of developing osteoarthritis increases with age.
Some of the risk factors that contribute to the development of osteoarthritis include:
Age (over 60 years)
Genetics
Being a woman
Being overweight
Have a pre-existing knee injury
Overuse of the knee or hip joint
Muscle weakness around the joint
Does osteoarthritis always get worse?
Osteoarthritis is a progressive condition that keeps getting worse if nothing is done to reduce pressure and wear on the joint.
Joint wear and tear that causes inflammation, swelling and stiffness leads to more weakness, further breakdown, more stiffness and more disability. The cycle continues if no change is made to break the cycle.
Although there is no cure for osteoarthritis, it is possible to slow or stop the progression of osteoarthritis through appropriate interventions…and most people don’t realize this!
Someone with osteoarthritis may experience periods of ‘flare-ups’, where the joint suddenly becomes swollen, stiff and/or painful. There may also be periods when symptoms disappear, range of motion is restored, and the joint feels relatively normal.
Controlling the “controllable factors” is an important tactic in managing osteoarthritis so that flare-ups don’t become worse than they need to be. Knowing what to do to calm a flare-up is an important step in long-term management and will help you continue doing the activities and sports you love.
Normal range of motion (ROM) helps spread the forces on the joint; Good STRENGTH helps support the joint; and good MOVEMENT PATTERNS help reduce abnormal forces on the joint. These concepts form the basis of what will SLOW or STOP the worsening of degenerative joint disease.
Losing weight if you are overweight, strengthening the muscles around the hip and knee, maintaining mobility and flexibility, and controlling inflammation and pain are important strategies to prevent osteoarthritis from worsening.
Common treatments for osteoarthritis
Activity
While it may seem like activity may initially cause more pain and swelling, physical activity has actually been proven to reduce symptoms and improve the quality of life for people with degenerative joint disease. Exercise promotes circulation and can lead to more pain-free days. Of course, you need to make sure you choose the right activity, progress the right way, and listen to your body.
A great place to start is by improving mobility and flexibility with simple stretching exercises.
Physiotherapy
Physical therapy has been proven to relieve symptoms, increase activity tolerance and improve the quality of life for people with osteoarthritis using a structured approach to restore range of motion, functional strength and balance. If you want to live an active lifestyle, exercise and enjoy the outdoors, ACL STRONG provides an expert training progression to strengthen your legs and core, improve balance and body control, and learn how to benefit your body and lifestyle in the long term. can protect. This is an online program and an excellent option if you want the convenience and freedom of exercising independently at home, while still receiving expert guidance and support.
Injectable products
Your doctor may be willing to try hyaluronic acid which can be injected into the affected arthritic joint. This fluid is similar in makeup to your body’s synovial fluid, which is like oil in your car. An injectable can lubricate affected bone and joint tissue to reduce the symptoms of osteoarthritis, making you feel more comfortable and delaying the need for surgery.
Arthroscopy
Arthroscopic surgery is an option when an orthopedic surgeon is confident that symptoms can be reduced by clearing out the surface tissue in the affected joint. Arthroscopic surgery is performed by placing small incisions in the joint to remove damaged tissue and create a smoother surface. The surgery will facilitate increased blood flow to the joint, which can help ‘restart’ the healing process and reduce the symptoms of osteoarthritis.
Joint replacement surgery
In cases where more conservative treatments have failed to relieve symptoms and improve quality of life, joint replacement surgery may become the best option. Replacing the degenerated joint surface with mechanical components can give you the recovery you need to be as active as you want.
Recovery from joint replacement surgery will involve several months of activity progression and physical therapy. Although the timeline varies for everyone, most patients return to normal activities and exercise, such as walking, biking, golfing, tennis, weight lifting, housework, and yard work. With hard work, dedication and consistency, many can return to their previous sports and activities at a higher level.
Osteoarthritis should NOT be daunting or intimidating. Receiving a diagnosis of osteoarthritis does not have to be confusing.
👉 How you deal with your condition is up to you. You CAN lead an active, healthy and sporty lifestyle, with the presence of degenerative joint diseases. Manage it with exercise, nutrition, making good choices and expert support, and you can continue living the lifestyle you choose. 💪
This was a descriptive, retrospective claims database study using the DATASUS database. The study identified patients with RA who sought care within SUS between January 1, 2010 and December 31, 2020.
The administrative claims data in DATASUS is presented as procedure codes from billing data and includes demographic information, all procedures (inpatient and outpatient), costs, and additional information23. Hospital admission (SIH [Sistema de Informações Hospitalares])24 and outpatient (SIA [Sistema de Informações Ambulatoriais])11 data exists separately and is linked at the patient level through multiple steps with different combinations of individual level information (date of birth, gender, city and zip code) for a probabilistic linkage approach. This Brazilian Healthcare Record Linkage (BRHC-RLK) methodology has been used in previous studies to enable a more comprehensive recording of each patient’s health record and thus allow a more complete evaluation of their journey through the SUS system.25. The method is based on multiple steps with different combinations of patient information from both databases, making it possible to identify or link patient data in both systems, while maintaining the anonymized nature of the database.
Patient-level data within DATASUS is anonymized and encrypted before being made available to researchers. DATASUS is publicly available and does not require further approval from ethics committees, according to Brazilian Ethics Resolution No. 510/2016.
Study population
The study population included patients with at least one claim of RA (according to the International Classification of Disease, 10th edition). [ICD-10] codes: M05.0, M05.3, M05.8, M06.0, M06.8, or M08.0) and ≥ 2 claims for disease-modifying antirheumatic drugs (DMARDs) ≥ 1 month apart in the 2010 survey– 2020 period. This study examined a treated population where the index date was the first DMARD claim and followed until the end of the study period (December 31, 2020) or the last available information. Detailed DMARD definitions can be found in Supplementary Table S1.
To capture initial treatment and address the potential for misclassification common in claims data, patients with a DMARD claim without an RA ICD-10 code 12 months prior to the index date were excluded. The index date was defined as the date of the first RA ICD-10 and DMARD prescription in the public health system during the study period. Patients with RA with less than six months of follow-up were excluded, in an effort to reduce the number of individuals with a false diagnosis or lack of follow-up in the database.
Because SUS is a healthcare system with universal coverage, patients with additional private health insurance can also receive medications (such as expensive drugs) covered by SUS at no out-of-pocket cost. This is often observed in other therapeutic areas26. For this reason, we stratified the results across the following cohorts: Cohort 1 is the entire study population, Cohort 2 is SUS-exclusive (i.e., dependent on SUS for all healthcare-related encounters, procedures, and treatments), and Cohort 3 represents SUS + private patients ( i.e. depending only on SUS for prescription drug coverage)26.
Measurements of DMARD treatment
DMARD treatments measured using procedure codes (see Supplementary Table S1) were grouped into the following categories: csDMARD for conventional synthetics and/or immunosuppressants (ciclosporin, cyclophosphamide, chloroquine, hydroxychloroquine, leflunomide, methotrexate, azathioprine and sulfasalazine), bDMARD for biological drugs (adalimumab, abatacept, etanercept, infliximab, rituximab, tocilizumab, golimumab, certolizumab) and tsDMARD for a synthetic, oral target therapy [Janus kinase (JAK) inhibitor, tofacitinib].
Treatment patterns were evaluated by specific drug (independently of monotherapy or in combination) as provided for RA treatment and the order of available treatments in SUS, by line of therapy (LOT), time point of each drug, previous and subsequent DMARD treatments in SUS. The first treatment was the first therapy from the admission according to RA ICD-10 code. LOT was defined as at least three claims (dispensation) of the same drug (b/tsDMARD) in a row. A new series of at least three claims (dispensation) of the same drug in the correct order was considered a new line of treatment. Thus, the switch to a treatment was identified as at least three claims for drugs other than the previous one, which are not included in the definition of drugs used in combination. Gaps were allowed regardless of time and did not constitute a new LOT. First-line (LOT1) refers to initial treatment, first b/tsDMARD claim of RA during the study period. Second-line (LOT2) refers to the second b/tsDMARD used for RA treatment, when the first b/tsDMARD was stopped. Third line (LOT3) refers to the third b/tsDMARD, when the previous b/tsDMARD was terminated. csDMARDs used before b/tsDMARD were also assessed. Switching treatment was defined as at least three claims for drugs different from the previous one (new LOT), and not part of drugs used in combination.
static analysis
Derived variables included age and distance to the clinic. Age was defined as the age at the first claim of an ICD-10 code for RA in the database. Distance was calculated as the Euclidean distance (km) between two zip codes: the patient’s place of residence and the health care facility or tomography or antiangiogenic treatment facility, as applicable. Treatment switch, discontinuation and end of follow-up were the main outcomes of censoring events of interest, also relevant in defining LOT and creating Sankey diagrams.
Continuous variables (e.g., age) are summarized by central tendency (means, medians) and dispersion (variance, range); and for categorical variables (e.g. gender) based on absolute number and percentage. Stratifications and/or sensitivity analyzes were performed to evaluate differences in gender, age groups, patient region of residence, drug use, treatment line, and others.
Stratified analyzes for mainstream and new users were prespecified, and for SUS-exclusive and SUS+ private cohorts. Frequent users were patients with RA who were currently receiving bDMARD treatment, and new users were patients with RA who were starting a new bDMARD treatment (i.e., their first prescription). To describe the use and sequential patterns of RA bDMARD treatments, patients were stratified by treatment type, LOT-specific drug, and SUS-exclusive status.
In multivariable logistic regression analyses, age, SUS-exclusive status, distance to clinic (160+ km), and pre-index cs/imsDMARD and other independent predictors were included to evaluate initiated therapy (LOT1) with b/tsDMARD (JAKi). Multivariable analyzes were performed using Cox regression models evaluating predictors by time to switch to tsDMARD (JAKi) compared to bDMARD (LOT2+), applying the same independent predictors from multivariable logistic regression analyzes (age, SUS-exclusive status , distance to clinic, pre-index cs/imsDMARD, other), plus the number of previously used bDMARDs. Sankey diagrams were used for visualizations of treatment patterns. Sankey diagrams quantitatively illustrate the sequence of treatment (and/or duration of treatment) and allow stratification by subpopulations of interest with censoring based on different treatment, discontinuation, or end of follow-up. Kaplan Meier survival analyzes and plots were generated for time to switch from LOT1 to LOT2, among those treated with b/tsDMARD, analyzed by drug type and by SUS-exclusive status.
The visual representation of the time-to-event of the switch from LOT1 to LOT2 in patients receiving b/tsDMARD therapies was presented in Kaplan-Meier curves. The last available patient information or end of study period was considered censored for patients who did not switch from LOT1 to LOT2.
All analyzes were performed using Python version 3.6.9 and statistical significance was set at p < 0.05.
Mighty Bone Loving Oatmeal contains a delicious nutritional boost. Oats are high in soluble fiber called beta-glucan, which is beneficial for heart health. They are a good source of manganese, a component of antioxidant enzymes, important in facilitating bone development and helping to make and break down glucose. Oats are also a good source of calcium, iron, magnesium, zinc and selenium.
Add berries
My bones feel nourished from eating my, cook once, twice (or 3-4 times), mighty, bone-loving oatmeal, especially as the weather turns colder. Because I find it so soothing, I usually eat oatmeal all year round. Adding berries rounds out this pleasant meal with a touch of sweetness.
Mighty Bone Loving Oatmeal
My mighty bone-loving oatmeal is not your everyday oatmeal. Check out the ingredients and enjoy a breakfast that’s both good for your bones and delicious.
Find more bone-loving recipes on my Comprehension Bone Health Program (CBHP) waiting list here!
View all recipes here.
Preparation time 1 dayD10 minutesminutes
Cooking time 20 minutesminutes
Total time 30 minutesminutes
Class Breakfast
kitchen American
Portions 4
Calories 339kcal
1/2cupsteel cut oatsbiological
1/2cupgritsorganic optional
1/4cupTeffoptional
1/4 cupQuinoa
1Tablespoonapple cider vinegarto soften
2/3cupwater
3Tablespoonchia seedsor Zen Basil Seeds
3Tablespoonflax seedground
1/2teaspoonspicescinnamon (ceylon is the best), nutmeg, cloves
1/2teaspoonvanilla extractoptional
1Tablespoonalmond butter
6plumsminced meat or 1 tablespoon plum puree
1handfulblueberriesoptional
1dashsalty
Soak oats, grits, teff and quinoa overnight in water with 1 tablespoon of apple cider vinegar. Make sure the grains are submerged in water. This starts the germination process.
In the morning, use a fine sieve to empty and rinse the soaked grains. You can also use a nut bag or cheesecloth bag.
Place the grains in a pot and add 3 cups of fresh water. Bring to the boil.
Add your spices and vanilla extract. If you are using chopped plums, add them now.
Reduce the temperature to medium heat
Stir your oatmeal
Add Zen Basil seeds or chia seeds
Cover your oatmeal and turn off the heat
Oatmeal should cook in about 20 minutes — you want it to be wet (but not runny), so when it’s time to add the ground flax, the oatmeal won’t be pasty.
The nutritional information is calculated automatically and may vary based on the ingredients and products used.
Below is the link to purchase mine downloadable guide:
Dr. Approved: 7-Day Meal Plan – Simple Cooking for Bone Health
With 27 bone-loving recipes, shopping lists, nutrition plans, storage tips, weekly tips (why soak), a nutrition panel with each recipe, 5 cooking videos for inspiration and much more.
My Fully Clickable (Table of Contents) download guide helps you navigate recipes and all the information in this guide.
The best $47 dollars you can spend on your bones.
Click the button below for more information and to make your purchase.
Post-injury outcomes following non-sports related concussion: a CARE Consortium study.
Roby PR, Mozel AE, Arbogast KB, Buckley T, Caccese JB, Chrisman SP, Clugston JR, Eckner JT, Esopenko C, Hunt T, Kelly LA, McDevitt J, Perkins SM, Putukian M, Susmarski A, Broglio SP, Pasquina PF , McAllister TW, McCrea M, Head CL; Researchers from the CARE consortium. J Athl train. September 8, 2023. doi: 10.4085/1062-6050-0181.23. E-publishing prior to printing. PMID: 37681681.
Full text freely available
Take home message
Athletes who sustain a non-sports-related concussion, compared to a sport-related concussion, are more likely to delay reporting, take longer to return to play, and have more symptoms.
Background
Most concussions are not related to sports, but most concussion research includes only sports-related concussions. When non-sports-related concussions are reported in the literature, participants come from a specialized concussion clinic or emergency department, potentially biasing the population toward those with more severe injuries or longer recoveries.
Study objectives
The authors examined the reporting characteristics and clinical outcomes of non-sports-related concussions among collegiate athletes.
Methods
The authors used data from the NCAA-DOD CARE Consortium. They grouped the participants based on the mechanism of their concussion: sports-related or non-sports-related. The authors only analyzed a person’s first concussion. Demographic information, injury information, and outcomes include, but are not limited to, age, gender, race, ethnicity, sport, history of concussion, the physician who made the initial diagnosis, time of reporting, delayed onset of symptoms, and a graduated symptom checklist. The authors also analyzed recovery outcomes, including duration of symptoms, provider who cleared the patient, and whether the patient experienced a slow recovery, which was defined as ≥14 days to asymptomatic or ≥24 days to indefinite return to play.
Results
The authors were able to analyze 3,500 people who suffered a concussion, 555 of which were non-sports related. Women suffered more than twice as many (23% versus 10%) non-sports-related concussions as men. The most common mechanisms of non-sports-related concussions were falls, slips or trips (28%), being hit by an object (26%), car accidents (20%) and unintentional contact with another person (9%). Patients who suffered a non-sports related concussion were less likely to report it immediately. They were four times more likely to require hospital transport, had greater severity of symptoms, longer duration of symptoms and more days lost due to injury. Athletes with a non-sports-related concussion were 1.5 times more likely to have a slow recovery and a greater chance of being diagnosed and cleared by a primary care physician than athletes with a sports-related concussion.
Viewpoints
Providers should keep in mind that patients who sustain a non-sports-related concussion are more likely to delay reporting, have a higher symptom burden, and take longer to recover. Athletic trainers must appreciate the unique challenges a patient with a non-sports-related concussion experiences in order to provide the same level of high-quality care.
Clinical implications
Sports medicine personnel can serve as a resource and advocate for education and referrals for patients who sustain a non-sports-related concussion. It is also important to emphasize to athletes the importance of reporting all concussions immediately, regardless of whether they occur during sport.
Related posts
Don’t wait any longer and report your concussion today CDC HEADS UP program increases concussion knowledge and injury communication
Written by Mitch Barnhart Reviewed by Jeffrey Driban
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Using more than one medication increases the chance of side effects, drug interactions, and the risk of falls. Learn ways to make sure your medications don’t increase your risk of falls. https://bit.ly/FPAW2023-Review-Medications-with-Your-Doctor
You may feel unsteady or start to fear falling as you get older. While that may be the case, there are exercise programs that can help improve balance, strength and flexibility. Find out how to stay strong and find an exercise program near you. https://bit.ly/FPAW2023-Find-A-Good-Exercise-Program
Each year, many older adults fall ill at home due to common household risks. Find out how to keep your home safe. https://bit.ly/FPAW2023-Maak-uw-huis-veilig
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If you’re working your way through the ACL prehab process, you may have heard a few ACL prehab goals to aim for before your doctor’s surgery. These goals often include goals such as:
Perform 20 straight leg movements without deceleration
Minimal effusion/swelling
Achieving these goals WILL put you in a much better position than if you avoided prehab altogether. However, much more can be accomplished during the prehab phase. The overarching goal we recommend is to get as close to 100% as possible before surgery (achieving the goals listed above is certainly not an indication that you are 100%). Doing so will increase your chances of optimal posterior recovery, increase your confidence toward surgery (which also plays a role in recovery), and potentially allow you to have a conversation with your doctor about ask whether or not you need surgery. (another blog post will go into that in more detail).
4 sleep tips to try
I am Dr. Susan E. Brown. I am a clinical nutritionist, medical anthropologist, writer and motivational coach speaker. Learn my proven 6-step natural approach to 
